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Its mechanism of action is currently being studied in large-scale clinical trials to evaluate both safety and long-term efficacy

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However, further preclinical studies, especially those focusing on ALD and MetALD models, and prospective clinical trials are necessary to fully translate these promising anticancer benefits to human patients

The R311C mutation results in a catastrophic loss-of-function as it destabilises key electrostatic interactions within the catalytic site that are essential for CaMKK2 activity [144]

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