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These nanovesicles act as high-speed delivery messengers

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Drug-Induced Anemia A reduction in red blood cells, which can

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Mechanistic investigation revealed ChaC1/AUR-driven cytotoxicity was attenuated by disulfide reducing agents (e.g., NAC and TCEP), while maintaining resistance to canonical programmed death pathway inhibitors targeting apoptosis, necroptosis, and ferroptosis
