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[DOI] [PubMed] [Google Scholar] 72.Balenovic D., Bencic M.L., Udovicic M., Simonji K., Hanzevacki J.S., Barisic I., Kranjcevic S., Prkacin I., Coric V., Brcic L., et al

[DOI] [PMC free article] [PubMed] [Google Scholar] El-Moselhy M

Interferon- Interferon- (IFN-) is mainly involved in the regulation of inflammatory immune responses, and levels are elevated in the intestinal mucosa in patients with IBD.39 Recent studies have also demonstrated that IFN- increases paracellular permeability in intestinal epithelial cells through the redistribution and expression of TJ proteins and the rearrangement of the actin cytoskeleton.40 Thus, IFN- increases actinmyosin contractility in a Rho-associated kinase-dependent manner and induces TJ protein internalization, resulting in intestinal TJ disruption

their hyperthyroid state may be induced by supplementation of thyroid hormones such as thyroxine (T4) or triiodothyronine (T3), or by the administration of thyroid-stimulating immunoglobulins to induce Graves disease, one of the autoimmune forms of hyperthyroidism (Zhang et al