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Even though APOE isoforms did not cause changes in the expression levels of major glucose transporters of the brain, GLUT1, GLUT3 and GLUT4 [95], APOE can indirectly alter glucose uptake in an isoform-dependent manner

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Moreover, when PKC is overexpressed, it can exacerbate oxidative stress by stimulating NADPH-oxidases and lipoxygenases, which are enzymes known to generate ROS, as evidenced in human platelet suspensions [67]