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This mechanism is especially important in disorders of mitochondrial fatty acids oxidation, when fatty acyl-CoAs accumulate in organs such as the heart inducing apoptosis and inflammation [5]

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Mitochondrial dysfunctions in ASD individuals could also result from various risk factors, both endogenous and exogenous, including toxins, immune stimulation, drugs, and metabolic abnormalities [17]

Two forms of VLCADD have been identified: an early (severe) form, which, if not recognised and diagnosed, can lead to cardiomyopathy and be life-threatening

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