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effects mediated mainly via ERR in cardiomyocytes
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The copper chelation capacity of AHK-Cu distinguishes it from unchelated peptide analogs, enabling research into coppers role as a cofactor in angiogenic and wound-response signaling
Gently turn the vial, without shaking it vigorously
The principle of using mAbs to increase IgG catabolism was also demonstrated in NHPs, when Nixon and colleagues at Dyax Corp used phage-display selected human IgG1 mAbs with high pH-independent affinity towards FcRn to reduce circulatory IgG without affecting albumin levels in NHPs [122]
