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While pharmacological inhibition of HDACs has generally been associated with enhanced cognitive function [6, 127], our results reveal a distinct, isoform-specific role for HDAC9: neuronal overexpression of HDAC9 preserves cognitive function during aging and mitigates AD-associated cognitive deficits and neuropathology

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Besides that, repeated administration of GA produces an antidepressant effect which has already been reported (Samad et al