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We set out to generate mosaic nanoparticles that co-display different E2E1-I53-50A trimers, in particular ones from different genotypes and with divergent HVR1 domains

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IL-1, IL-6 and TNF- promote the transcription of MICA/B and PD-L1 gene via PI3K/AKT, JAK/STAT3, and MKK/p38 MAPK pathways To identify the mechanisms by which pro-inflammatory cytokines modulate MICA/B and PD-L1, we first evaluated the transcription levels of MICA, MICB, and PD-L1