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Loss of FXR or Bile Acid-dependent Inhibition accelerate carcinogenesis of Gastroesophageal Adenocarcinoma

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Given the mere mass of malignant cells within a solid tumor such as HCC, we hypothesized that tumor targeting and efficient killing of tumor cells would require a stable expression of an HBV-specific TCR to allow T cells to expand while maintaining their specificity and effector function

For certain treatments, the full effects may become more noticeable within a few hours or by the next day.
