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Mantovani, G., Maccio, A., Madeddu, C., Gramignano, G., Serpe, R., Massa, E., Dessi, M., Tanca, F

Key actions include: Activation of the folate-polyamine-AICAR pathway, triggering AMPK signaling independently of cellular energy depletion Enhanced glucose uptake in skeletal muscle via GLUT4 translocation, resembling insulin-responsive metabolic pathways Interaction with nuclear transcription factors to support antioxidant defense and control of inflammatory pathways Through these mechanisms, MOTS-c functions as a mitochondria-to-nucleus messenger, facilitating cellular adaptation to energy demands and oxidative stress

[DOI] [PubMed] [Google Scholar] 91.Chernyavsky A.I., Galitovskiy V., Shchepotin I.B., Jester J.V., Grando S.A

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doi: 10.1016/j.biopha.2020.110714