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The exact mechanism of eNOS modification is unknown but in vivo experiments have shown that eNOS activation in aortas and iNOS transgenetic expression in mouse heart both result in NO-induced protein S-glutathionylation

2015), gene therapy using AAV vectors to deliver glutamate decarboxylase (GAD) into the STN has been introduced (Fig

1D and Supplementary Fig

Elabela treatment reduced ferroptosis and improved myocardial hypertrophy and remodeling in hypertensive mice by normalizing the levels of xCT and GPX4 [96]

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[DOI] [PMC free article] [PubMed] [Google Scholar] Roberts BW, Kilgannon JH, Hunter BR, Puskarich MA, Pierce L, Donnino M, Leary M, Kline JA, Jones AE, Shapiro NI, Abella BS, Trzeciak S, 2018